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Registro completo
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Biblioteca (s) : |
INIA La Estanzuela. |
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Fecha : |
10/05/2021 |
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Actualizado : |
10/05/2021 |
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Tipo de producción científica : |
Abstracts/Resúmenes |
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Autor : |
ROMAN, L.; BANCHERO, G.; MORALES-PIÑEYRUA, J.; ACOSTA, Y.; MENDOZA, A.; PLA, M.; LA MANNA, A. |
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Afiliación : |
LORENA CAROLINA ROMAN GAY, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; GEORGGET ELIZABETH BANCHERO HUNZIKER, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; JESSICA TATIANA MORALES PIÑEYRUA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; YAMANDU MARTIN ACOSTA AZPIROZ, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; ALEJANDRO FRANCISCO MENDOZA AGUIAR, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; MARCELO PLA TEJERA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; ALEJANDRO FRANCISCO LA MANNA ALONSO, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
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Título : |
Efecto del acceso a sombra artificial durante el periodo seco de vacas lecheras.[Effect of access to artificial shade during the dry period in dairy cows]. |
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Complemento del título : |
SP 52. |
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Fecha de publicación : |
2015 |
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Fuente / Imprenta : |
In: Congreso Argentino de Producción Animal, 38., 2015. Resúmenes. Santa Rosa, La Pampa, AR: ASAS/AAPA, 2015. |
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Páginas : |
p. 134. |
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Serie : |
(Revista Argentina de Producción Animal; 2015; 35; supl.1). |
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Idioma : |
Español |
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Contenido : |
Conclusiones: El acceso a sombra artificial durante los últimos 60 días de gestación no afecto la TR, el PV y la CC animal, mientras
que mostró efecto sobre la FR a las 13:30 y 17 horas. Se observó efecto sobre el consumo de alimento preparto sin
afectar el consumo posparto y el consumo de agua. |
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Palabras claves : |
PERÍODO SECO. |
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Thesagro : |
ESTRES TERMICO; LECHERÍA; VACA LECHERA. |
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Asunto categoría : |
-- |
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URL : |
http://www.ainfo.inia.uy/digital/bitstream/item/15574/1/Revista-Argentina-de-Produccion-Animal-2015.v.35.supl.1.-SP-52.pdf
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Marc : |
LEADER 01182nam a2200253 a 4500
001 1062052
005 2021-05-10
008 2015 bl uuuu u01u1 u #d
100 1 $aROMAN, L.
245 $aEfecto del acceso a sombra artificial durante el periodo seco de vacas lecheras.[Effect of access to artificial shade during the dry period in dairy cows].$h[electronic resource]
260 $aIn: Congreso Argentino de Producción Animal, 38., 2015. Resúmenes. Santa Rosa, La Pampa, AR: ASAS/AAPA$c2015
300 $ap. 134.
490 $a(Revista Argentina de Producción Animal; 2015; 35; supl.1).
520 $aConclusiones: El acceso a sombra artificial durante los últimos 60 días de gestación no afecto la TR, el PV y la CC animal, mientras que mostró efecto sobre la FR a las 13:30 y 17 horas. Se observó efecto sobre el consumo de alimento preparto sin afectar el consumo posparto y el consumo de agua.
650 $aESTRES TERMICO
650 $aLECHERÍA
650 $aVACA LECHERA
653 $aPERÍODO SECO
700 1 $aBANCHERO, G.
700 1 $aMORALES-PIÑEYRUA, J.
700 1 $aACOSTA, Y.
700 1 $aMENDOZA, A.
700 1 $aPLA, M.
700 1 $aLA MANNA, A.
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INIA La Estanzuela (LE) |
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 | Acceso al texto completo restringido a Biblioteca INIA La Estanzuela. Por información adicional contacte bib_le@inia.org.uy. |
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Registro completo
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Biblioteca (s) : |
INIA La Estanzuela. |
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Fecha actual : |
02/03/2017 |
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Actualizado : |
07/03/2017 |
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Autor : |
GARCIA, J.P; GIANNITTI, F.; FINNIE, J.W.; MANAVIS, J. .; BEINGESSER, J.; ADAMS ,V.; ROOD, J.I.; UZAL, F.A. |
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Afiliación : |
FEDERICO GIANNITTI, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
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Título : |
Comparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives. |
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Fecha de publicación : |
2015 |
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Fuente / Imprenta : |
Veterinary Pathology, 2015, v.52.n.6. p.1250-1253, 2015. |
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Idioma : |
Inglés |
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Contenido : |
Abstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease. MenosAbstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated wi... Presentar Todo |
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Thesagro : |
ENFERMEDADES DE LOS ANIMALES. |
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Asunto categoría : |
-- |
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Marc : |
LEADER 02512naa a2200217 a 4500
001 1056750
005 2017-03-07
008 2015 bl uuuu u00u1 u #d
100 1 $aGARCIA, J.P
245 $aComparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives.$h[electronic resource]
260 $c2015
520 $aAbstract Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease.
650 $aENFERMEDADES DE LOS ANIMALES
700 1 $aGIANNITTI, F.
700 1 $aFINNIE, J.W.
700 1 $aMANAVIS, J. .
700 1 $aBEINGESSER, J.
700 1 $aADAMS ,V.
700 1 $aROOD, J.I.
700 1 $aUZAL, F.A.
773 $tVeterinary Pathology, 2015$gv.52.n.6. p.1250-1253, 2015.
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